@article{oai:nagasaki-u.repo.nii.ac.jp:00001582,
 author = {TOKUYAMA, Shogo and TAKAHASHI, Masakatsu and KANETO, Hiroshi},
 issue = {6},
 journal = {Journal of Pharmacobio-Dynamics},
 month = {},
 note = {In mice, both swimming-stress induced analgesia (SW-SIA) and clonidine (CLO) analgesia were dose dependently antagonized by yohimbine, an α2-adrenoceptor antagonist, but not by naloxone, an opioid μ-antagonist. SW-SIA was potentiated by subanalgesic dose of CLO, and CLO analgesia was enhanced by SW-SIA. Animals tolerant to CLO analgesia were tolerant to SW-SIA, in contrast, CLO analgesia was potentiated in SW-SIA tolerant mice. Thus, SW-SIA and CLO analgesia partially share a common α2-adrenergic-dependent mechanism, for their production., Journal of Pharmacobio-Dynamics, 14(6), pp.357-361; 1991},
 pages = {357--361},
 title = {Participation of an α2-mediated mechanism in the production of forced swimming-stress induced analgesia in mice.},
 volume = {14},
 year = {1991}
}