@article{oai:nagasaki-u.repo.nii.ac.jp:00022065,
 author = {Kinoshita, Ikuo and Motomura, Masakatsu and Nagasato, Kunihiko and Ichinose, Katsuhiro and Ohishi, Kiyosumi and Takeo, Gou and Satoh, Akira and Nakamura, Tatsufumi and Yoshimura, Toshiro and Tsujihata, Mitsuhiro and Nagataki, Shigenobu},
 issue = {1-3},
 journal = {Acta medica Nagasakiensia},
 month = {Oct},
 note = {We measured antiphospholipid antibodies in sera from 94 patients with myasthenia gravis (MG). We found lgG aCL in 14/94 (14.9 % ), lgM aCL in 6/94 (6.4 %) and LA in 4/56 (7.1 %) patients with MG. As a whole 21 of 94 (22.3 % ) patients with MG had some aPL. There was no correlation between the presence of aPL and the severity of MG, the presence of hyperplasia of thymus, titer of the antiacetylcholine receptor antibodies or anti-single stranded DNA antibodies. Though the percentage of malignant thymoma with aPL were higher than that of malignant thymoma without aPL, we thought that aPL were not the specific antibody in malignant thymoma. In MG, aPL did not play as the aCL syndrome and seemed to be non-specific antibodies., Acta medica Nagasakiensia. 1994, 39(1-3), p.41-44},
 pages = {41--44},
 title = {Antiphospholipid Antibodies in Patients with Myasthenia Gravis},
 volume = {39},
 year = {1994}
}